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DHEA mechanism as antidepressant may act via modulation with the GABAARs activity. The GABA ergic system can mediate depression (Lloyd et alibi, 1989). In depressed patients, cerebral spinal fluid amounts of GABA are below in healthy controls (Gerner and Hare, 1981; Gold et alibi, 1980). Furthermore, low GABA content is correlated with depression in post mortem temporal and frontal cortices from patients with Alzheimer’s disease (Garcia Alloza et aliae, 2006) at www.07runescapegolds.com.au .
Through these patients, a higher density of GABAARs correlates with all the severity of depression. Rapidly increased cortical power of GABA upon antidepressant management of healthy individuals (Bhagwagar et aliae, 2004) and electroconvulsive therapy for depressed patients (Sanacora et aliae, 2003), anti depressants that affect GABA haven’t yet been developed. The subunit composition determines the pharmacological properties of GABAAR. Drugs, including neurosteroids, which connect to the receptors, can affect the subunit mRNA expression (Sun et al., 2004). GABAAR usually contains two two and either a or using the being rare (Sun et alibi, 2004). GABAARs containing an extremely than the usual are definitely more sensitive to neurosteroids (Belelli et alii, 2002), and mice lacking GABAA are less understanding of neurosteroids (Brown et al, 2002;